This study shows that “hyperactive” enteric glia contribute to mechanisms that drive long-lasting changes in gut motility following an acute bout of inflammation through abnormal neuron-to-glia and glia-to-neuron communication.
Hyperactive enteric glia contribute to persistent dysmotility following inflammation by driving aberrant excitatory responses in neurons / Seguella, Luisa; Thomasi, Beatriz; Basili Franzin, Silvia; L Mcclain, Jonathon; Lavalle, Rafaella; Zilli, Aurora; Parmar, Visha; Esposito, Giuseppe; D Gulbransen, Brian. - In: CMGH. - ISSN 2352-345X. - 20:2(2025), pp. 1-23. [10.1016/j.jcmgh.2025.101634]
Hyperactive enteric glia contribute to persistent dysmotility following inflammation by driving aberrant excitatory responses in neurons
Luisa Seguella;Silvia Basili Franzin;Aurora Zilli;Giuseppe Esposito;
2025
Abstract
This study shows that “hyperactive” enteric glia contribute to mechanisms that drive long-lasting changes in gut motility following an acute bout of inflammation through abnormal neuron-to-glia and glia-to-neuron communication.File allegati a questo prodotto
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