An increase of the latency of N1 and P100 components at the highest spatial frequency supports the theory that in juvenile diabetes, without any ocular sign of disease, a subclinical functional alteration of the foveal area is present: the lathency of the N1 component seemsto be more sensible to the long-term metabolic condition and the difference with that of the P100 component support the hypothesis of a different origin of these two components.

Pattern VEPs and long-term metabolic control (HbAlc) in juvenile diabetes . Source: INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE Volume: 37 Issue: 3 Pages: 4448-4448 Published: FEB 15 1996 Times Cited: 0 (from Web of Science) / Rispoli, Eduardo; Tommasini, P; Perdicchi, A; De Leo A., Moauro e; Malagola, Romualdo; Pannarale, Luigi. - In: INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE. - ISSN 0146-0404. - STAMPA. - Volume: 37 Issue: 3:(1996), pp. 4448-4448. (Intervento presentato al convegno ARVO 1996 tenutosi a FORT LAUDERDALE FLORIDA nel 1-5 MAGGIO 1996).

Pattern VEPs and long-term metabolic control (HbAlc) in juvenile diabetes . Source: INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE Volume: 37 Issue: 3 Pages: 4448-4448 Published: FEB 15 1996 Times Cited: 0 (from Web of Science)

RISPOLI, Eduardo;MALAGOLA, Romualdo;PANNARALE, Luigi
1996

Abstract

An increase of the latency of N1 and P100 components at the highest spatial frequency supports the theory that in juvenile diabetes, without any ocular sign of disease, a subclinical functional alteration of the foveal area is present: the lathency of the N1 component seemsto be more sensible to the long-term metabolic condition and the difference with that of the P100 component support the hypothesis of a different origin of these two components.
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11573/422757
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